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A significant body of evidence has highlighted the role of cytokines in the regulation of various tight junction proteins in a multitude of pathological conditions

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When SAM is abundant, indicative of high methionine levels, SAM inhibits the enzymes methylenetetrahydrofolate reductase (MTHFR) and BHMT to limit conversion of homocysteine to methionine, allowing homocysteine to be diverted for transsulfuration [5,6]