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As shown in Figures 2E, D YRK1A expression in HepG2 cells induced a significant increase in H 2 S production without affecting cell proliferation and/or viability (Supplementary Figure 1C), whereas two mutant forms of DYRK1A (p.S324R and p.S311F) that had lost their kinase activity (Courraud et al., 2021) had no effect on H 2 S production (Figure 2E)

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