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Newer, indirect evidence includes the antibiotic-induced switching to anaerobic metabolism, so that ROS production is reduced [12], and the restoration of carbapenem susceptibility of carbapenemase-producing Escherichia coli by adding oxidized glutathione, which impairs the antioxidant status of the bacterial cell [13]

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Furthermore, the mutation status of the BRAF gene and the use of BRAF inhibitors both influence the oxidative metabolism and oxidative phosphorylation levels of melanoma cells, potentially determining their sensitivity to ferroptosis inducers (185)

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