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Metabolic pathways of glutathione synthesis, recycling,... | Download Scientific Diagram

doi: 10.1055/s-2007-981457

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Accordingly, a strong positive correlation was detected between the proportion of cells in a population that entered senescence after DNA damage and the species’ lifespan ( Mitochondria-derived oxidative stress together with genomic and mtDNA mutations drive progressive mitochondrial dysfunction with age, including increased proton leak, impaired cardiolipin-dependent protein function, and diminished ATP production, which together lead to widespread cellular deterioration and implicate mitochondrial damage as an important self-amplifying contributor to organismal aging (Shigenaga et al., 1994)