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Consistently, in vitro studies have shown that silencing TXNIP reduces autophagic vesicle accumulation and the expression of LC3-II and p62 in renal tubular cells exposed to high glucose, thereby enhancing autophagic flux and resolving autophagic dysfunction (150)
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In a rodent model of obstructive sleep apnea-hypopnea, effects of SF intervention were examined [274]
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