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SIRT1 targets over 100 different proteins involved in metabolism, stress responses, and longevity pathways, including the tumor suppressor p53, the transcriptional coactivator PGC-1α, and the DNA repair protein Ku70. Through deacetylation of PGC-1α, SIRT1 promotes mitochondrial biogenesis and oxidative metabolism, while deacetylation of p53 reduces apoptotic responses to mild stress, promoting cellular survival and longevity
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The hypothesis that intracellular metal availabilities will be set to the opposite of the Irving-Williams series is not new [217]
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