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The studies began with the determination of the optimal dosing for Cagrilintide in both chow-fed and high-fat diet (HFD)-fed wild-type (WT) mice, followed by analyses of its receptor dependence using receptor activity-modifying protein (RAMP) knockout (KO) models, and mechanistic studies involving neuronal activation and gene expression profiling [2]

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The level of sulfane sulfur-containing compounds in the MPS cell lines (1% DMSO) was reduced regardless of whether the culture medium contained serum or not (the WT and Naglu −/− cells), and regardless of how DMSO was introduced into the cells (the Naglu −/− cells) (Table 1)