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Heterogeneity of growth hormone-dependent lipolytic factors

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Peter calibrates his enthusiasm to the strength of the evidence, and usually he’s NOT overly enthusiastic about peptides in this category CJC-1295 is a useful example It is both biologically plausible and biologically active Also, for what it’s worth, it’s probably one of the 3 or 4 peptides Peter gets asked about the most Again, we’ve covered this in much greater detail in the previous AMA if you want to go back and get really into the weeds of the history of this peptide and all those other things ⇒ CJC-1295 can raise growth hormone and IGF-1 “ One of the biggest mistakes people make is assuming that’s because a molecule changes biology, therefore it’s going to improve health .”‒ Peter Attia Almost every drug we study changes biology in some way If it binds to a receptor, alters a signaling pathway, raises one biomarker, lowers another, or changes the expression of a protein, that’s changing biology That’s not the relevant question The question is whether or not those biological changes translate into outcomes that actually matter to patients Better function, more strength, faster healing, less pain, fewer heart attacks, longer life, or measurable improvement in quality of life That distinction matters a lot for CJC-1295, because its appeal rests on stimulating the growth hormone pathway And we already have a much more direct way to interrogate that pathway, giving growth hormone itself In people who are truly growth hormone deficient, replacement can matter In a few specific clinical conditions (such as HIV-associated lipodystrophy), targeting this pathway can have an incredible role But in growth hormone replete adults (i.e