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Deficiency in key autophagy molecules, such as Atg7 and Beclin-1, may contribute to DOR (Texada et al., 2019), as confirmed in animal studies

YGroup_all_duo

mg:5

However, glutathione was still significantly reduced with rapamycin treatment in cells expressing the rapamycin-resistant ATF4, suggesting additional ATF4-independent mechanisms contributing to this regulation (Lam et al., 2017)