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In summary, AS-IV negatively regulates fibroblast fibrosis, as primarily indicated by the remarkable promotion of the dephosphorylation of Smad2 and Smad3 in fibroblasts and restoration of Smad7 activity, indicating that AS-IV mediates positive feedback inhibition and the negative feedback enhancement of the TGF-β1/Smads signaling pathway (77,78)
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