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This suggests that ferroptosis contributes to placental dysfunction in RM (Xie et al., 2024)

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Studies utilizing in silico analyses, neuronal cultures, and animal models have identified key molecular targets, including genes such as prostaglandin-endoperoxide synthase 2 (PTGS2), glutathione reductase (GSR), and BDNF, as well as transcription factors like Nrf-2 and peroxisome proliferator-activated receptor (PPAR)