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PKC action or spontaneous glucose fragmentation into methylglyoxal and other small ketoaldehydes activate the AGE–RAGE pathway, which seems an effective way to promote the expression of NFκB and its pro-oxidant reinforcement
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Sontheimer) as an alternative to the “IIM” designation because it allows for the inclusion of these cutaneous subsets of DM1–16 (eTable 156-0.2)
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