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When drugs with the same or similar molecular structures are re-exposed to immunized individuals, they can immediately bind to the cross-linking specific IgE and, within minutes, stimulate the release of inflammatory mediators such as histamine and tryptase, and elicit the subsequent rapid generation of prostaglandins and cytokines, which cause a systemic inflammatory reaction (mainly in the skin and airways) such as urticaria, anaphylaxis, and asthma ( + and CD8 + T cells participate in type IV ADHRs, of which CD8 + T cells are considered to play a major role ( + T-cell responses

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30 (10), x3–x11