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Recent reports suggest cuprizone does not accumulate in the brain;[5] rather, cuprizone toxicity extensively modifies copper and zinc distribution in the brain, resulting in mitochondrial dysfunction that leads to demyelination.[6–9] Spatial heterogeneity in brain pathology in the cuprizone model has been demonstrated,[10–15] and the mechanism of demyelination may vary across structures
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[DOI] [PMC free article] [PubMed] [Google Scholar] 38.Duarte A., Martinho A., Luís Â., Figueiras A., Oleastro M., Domingues F.C., Silva F
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