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However, both preclinical 17–20 and clinical 21,22 studies largely agree that GIP and GLP-1 are the two primary physiological incretins that mediate nutrient signaling from the intestine to the pancreatic islets, to properly regulate insulin secretion in control of postprandial glucose metabolism
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Several comprehensive reviews have been published which described the types of proteins susceptible to glutathionylation and the effects of post-translational modifications on their functions in detail [3,80,85]
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