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Both mechanisms, result in persistent hyperactivity in neutrophils of periodontitis patients.[99] Activities of certain pathogens and cigarette smoking have shown to lower glutathione levels within periodontal cells, and OS depletes glutathione, resulting in activation of redox-sensitive transcription factors and thus in the creation of a pro-inflammatory state.[4] Certain putative periodontopathogens, e.g., Fusobacterium nucleatum vincentii, F

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Moreover, these models used adult microbiota, despite evidence that infants are found to contain higher levels of MPs in fecal samples (Zhang et al., 2021b)

cure or prevent any disease